PROFILE

NIYONSABA François, M.D., Ph.D.


Department of Medical Interpreting

Short Biography

1989-1994
M.D. China Medical University, Liaoning, China
1995-1998
Master’s Degree, Department of Orthopedics, China Medical University, Liaoning, China
1999-2003
Ph.D., Department of Host Defense and Biochemistry, Graduate School of Medicine, Juntendo University Graduate School of Medicine
2003-2006
Instructor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine
2006-2007
Assistant Professor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine
2007-2021
Associate Professor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine
2010-2011
Visiting Associate Professor, Department Microbiology and Immunology, University of British Columbia, Vancouver, Canada
2015-2017
Senior Associate Professor, Global Health Studies, Faculty of International Liberal Arts, Juntendo University
2017-Present
Professor, Global Health Studies, Faculty of International Liberal Arts, Juntendo University
2021-Present
Senior Associate Professor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine
 

Keywords

  • Antimicrobial peptides
  • Infections
  • Skin Barrier
  • Skin Immunology
  • Wound Healing

Main Research Topics and Interests

  • Roles of Antimicrobial Agents in the Skin Immunity (Graduate School of Medicine)
  • Global Infections (Faculty of International Liberal Arts)

Publications (in English)

  • Original Articles: 62
  • Review Articles: 18
  • Books: 3
  • h-index: 30 (Web of Science)
  • Sum of Times Cited: 3,769 (Web of Science)

Recent Main Publications

  • Antimicrobial peptide derived from insulin-like growth factor-binding protein 5 activates mast cells via Mas-related G protein-coupled receptor X2. Allergy 75(1): 203-207; 2020.
  • Involvement of the lipoprotein receptor LRP1 in AMP-IBP5-mediated migration and proliferation of human keratinocytes and fibroblasts. J Dermatol Sci 99(3): 158-167, 2020.
  • The antimicrobial peptide human β-defensin-3 accelerates wound healing by promoting angiogenesis, cell migration and proliferation through the FGFR/JAK2/STAT3 signaling pathway. Front Immunol 2021 (In press).
  • More

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