PROFILE

WATADA Hirotaka, M.D., Ph.D.


Department of Metabolism & Endocrinology

Short Biography

1990
M.D. Osaka University Faculty of Medicine
1996-1998
JSPS Fellow
1997
Ph.D. Osaka University Graduate School of Medicine
1997-2000
Post-Doctoral Fellow, Hormone Research Institute, University of California San Francisco
2001-2006
Assistant Professor, Department of Metabolism & Endocrinology, Juntendo University Faculty of Medicine
2006-2010
Associate Professor, Department of Metabolism & Endocrinology, Juntendo University Faculty of Medicine
2010-Present
Professor, Department of Metabolism & Endocrinology, Juntendo University Graduate School of Medicine
 

Keywords

  • Diabetes Mellitus
  • Pancreatic Islets
  • Atherosclerosis
  • Insulin Resistance in East Asian
  • Autophagy

Main Research Topics and Interests

  • Pancreatic beta cell development and regeneration.
  • Regulation of pancreatic beta cell mass
  • Physiology of insulin resistance in East Asian
  • Progression of atherosclerosis in diabetes
  • Clinical Diabetology

Publications (in English)

  • Original Articles: 387
  • Review Articles: 22
  • h-index: 58 (Web of Science)
  • Sum of Times Cited: 18,409 (Web of Science)

Recent Main Publications

  • Takahashi M, Miyatsuka T, Suzuki L, et al. Biphasic changes in beta-cell mass around parturition are accompanied by increased serotonin production. Sci Rep 2020;10:4962.
  • Kiya M, Tamura Y, Takeno K, et al. Adipose Insulin Resistance and Decreased Adiponectin Are Correlated With Metabolic Abnormalities in Nonobese Men. J Clin Endocrinol Metab. 2021 Apr 23;106(5):e2228-e2238
  • Wakasugi S, Mita T, Katakami N, et al. Associations between continuous glucose monitoring-derived metrics and arterial stiffness in Japanese patients with type 2 diabetes. Cardiovasc Diabetol. 2021 ;20(1):15.
  • Osonoi Y, Mita T, Azuma K, et al. Defective autophagy in vascular smooth muscle cells enhances cell death and atherosclerosis. Autophagy 2018;14:1991-2006.
  • Miura M, Miyatsuka T, Katahira T, et al. Suppression of STAT3 signaling promotes cellular reprogramming into insulin-producing cells induced by defined transcription factors. EBioMedicine 2018;36:358-366.
  • More

Other Specific Comments

  • Associate Editor of Journal of Clinical Endocrinology and Metabolism

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